In this paper, the model plant Arabidopsis thaliana wild type Col-0 and tyrosine degradation pathway defective mutant sscd1 were used as experimental materials to explore the regulatory mechanism of anthocyanin biosynthesis, to analyze the accumulation of anthocyanin and anthocyanin biosynthesis related gene expression after treatment with exogenous phenylalanine, and to explore whether the blocked tyrosine degradation pathway affect phenylalanine-induced anthocyanin accumulation. The results showed that the accumulation of anthocyanins in sscd1 mutant seedlings increased after phenylalanine treatment, meanwhile, the expression levels of anthocyanin biosynthetic genes including PAL, CHI, CHS, DFR, LDOX and UF3GT were significantly upregulated in sscd1 mutants. This results indicated that blocking tyrosine degradation by SSCD1 gene mutation could increase the synthesis of anthocyanin.