拟南芥psc1突变体降低蔗糖诱导的花青素积累
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国家自然科学基金项目(30770195)


The psc1 mutant reduces the accumulation of sucrose-inducedanthocyanin in Arabidopsis
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    摘要:

    以拟南芥野生型和psc1突变体为材料,研究蔗糖(30、60、90、120、150 mmol/L)诱导花青素的积累。结果表明:蔗糖浓度高于60 mmol/L时,拟南芥psc1突变体中花青素的积累比野生型明显降低,花青素生物合成关键基因DFR的表达量减少。在无蔗糖和有蔗糖(90 mmol/L)条件下,添加表油菜素内酯进一步分析拟南芥psc1突变体中花青素的积累,结果显示,表油菜素内酯能显著增加拟南芥psc1突变体中花青素含量以及DFR基因的表达。累。

    Abstract:

    Arabidopsis wild-type and psc1 mutant were used to investigate the anthocyanin accumulation induced by sucrose. The results showed that the anthocyanin was induced by sucrose and the expression of DFR, a key gene in anthocyanin biosynthesis, compared with wild-type, was clearly reduced in the psc1 mutant when the sucrose concentration was higher than 60 mmol/L. The psc1 mutant was treated by epibrassinolide with or without sucrose, and it was found that the application of epibrassinolide remarkably increased the sucrose-induced anthocyanin content and the expression of DFR gene of the psc1 mutant as well.

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袁利兵,张琨,彭志红,任春梅.拟南芥psc1突变体降低蔗糖诱导的花青素积累[J].湖南农业大学学报:自然科学版,2011,37(2):139-142.

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历史
  • 收稿日期:2010-09-26
  • 最后修改日期:2011-03-09
  • 录用日期:2011-03-31
  • 在线发布日期: 2011-04-12
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